FWC Proposes Rule Affecting Importation of Deer

<Photo : www.myfwc.com

The Florida Fish and Wildlife Conservation Commission (FWC) wants to hear from deer enthusiasts and those who have deer farms or hunting preserves about a proposed rule change that would prohibit the importation of live captive deer into Florida from out-of-state sources.

The change is being proposed in an effort to reduce the chances of chronic wasting disease (CWD) being introduced into the state. CWD is not known to affect people.

The disease, which has been discovered in 22 states, two Canadian provinces and in South Korea, is a contagious neurological disease affecting deer, elk and moose. It is similar to mad cow disease, always fatal, and there is no known cure or vaccine for the animals.

The rule-change proposal will go before the Commission at its June 13 meeting in Lakeland, and the FWC encourages all those who have an interest to go to MyFWC.com/Deer and select “Captive Cervids” to read about what is being proposed and offer any comments, questions or concerns they may have.

If the proposal is passed at the June meeting, it would go into effect soon after.
For more information on CWD, go to www.CWD-info.org.

  • flounder9

    smart move…

    Tuesday, April 16, 2013

    Cervid Industry Unites To Set Direction for CWD Reform and seem to ignore
    their ignorance and denial in their role in spreading Chronic Wasting


    Thursday, May 26, 2011

    Travel History, Hunting, and Venison Consumption Related to Prion Disease
    Exposure, 2006-2007 FoodNet Population Survey

    Journal of the American Dietetic Association Volume 111, Issue 6 , Pages
    858-863, June 2011.


    Saturday, February 18, 2012

    Occurrence, Transmission, and Zoonotic Potential of Chronic Wasting Disease

    CDC Volume 18, Number 3—March 2012


    Interspecies transmission of CWD to noncervids has not been observed under
    natural conditions. CWD infection of carcass scavengers such as raccoons,
    opossums, and coyotes was not observed in a recent study in Wisconsin (22). In
    addition, natural transmission of CWD to cattle has not been observed in
    experimentally controlled natural exposure studies or targeted surveillance (2).
    However, CWD has been experimentally transmitted to cattle, sheep, goats, mink,
    ferrets, voles, and mice by intracerebral inoculation (2,29,33).

    CWD is likely transmitted among mule, white-tailed deer, and elk without a
    major species barrier (1), and other members of the cervid family, including
    reindeer, caribou, and other species of deer worldwide, may be vulnerable to CWD
    infection. Black-tailed deer (a subspecies of mule deer) and European red deer
    (Cervus elaphus) are susceptible to CWD by natural routes of infection (1,34).
    Fallow deer (Dama dama) are susceptible to CWD by intracerebral inoculation
    (35). Continued study of CWD susceptibility in other cervids is of considerable

    Reasons for Caution There are several reasons for caution with respect to
    zoonotic and interspecies CWD transmission. First, there is strong evidence that

    distinct CWD strains exist (36). Prion strains are distinguished by varied
    incubation periods, clinical symptoms, PrPSc conformations, and CNS PrPSc
    depositions (3,32). Strains have been identified in other natural prion
    diseases, including scrapie, BSE, and CJD (3). Intraspecies and interspecies
    transmission of prions from CWD-positive deer and elk isolates resulted in
    identification of >2 strains of CWD in rodent models (36), indicating that
    CWD strains likely exist in cervids. However, nothing is currently known about
    natural distribution and prevalence of CWD strains. Currently, host range and
    pathogenicity vary with prion strain (28,37). Therefore, zoonotic potential of
    CWD may also vary with CWD strain. In addition, diversity in host (cervid) and
    target (e.g., human) genotypes further complicates definitive findings of
    zoonotic and interspecies transmission potentials of CWD.

    Intraspecies and interspecies passage of the CWD agent may also increase
    the risk for zoonotic CWD transmission. The CWD prion agent is undergoing serial
    passage naturally as the disease continues to emerge. In vitro and in vivo
    intraspecies transmission of the CWD agent yields PrPSc with an increased
    capacity to convert human PrPc to PrPSc (30). Interspecies prion transmission
    can alter CWD host range (38) and yield multiple novel prion strains (3,28). The
    potential for interspecies CWD transmission (by cohabitating mammals) will only
    increase as the disease spreads and CWD prions continue to be shed into the
    environment. This environmental passage itself may alter CWD prions or exert
    selective pressures on CWD strain mixtures by interactions with soil, which are
    known to vary with prion strain (25), or exposure to environmental or gut

    Given that prion disease in humans can be difficult to diagnose and the
    asymptomatic incubation period can last decades, continued research,
    epidemiologic surveillance, and caution in handling risky material remain
    prudent as CWD continues to spread and the opportunity for interspecies
    transmission increases. Otherwise, similar to what occurred in the United
    Kingdom after detection of variant CJD and its subsequent link to BSE, years of
    prevention could be lost if zoonotic transmission of CWD is subsequently
    identified, CWD will likely continue to emerge in North America. …




    Generation of a new form of human PrPSc in vitro by inter-species
    transmission from cervids prions

    Our results have far-reaching implications for human health, since they
    indicate that cervid PrPSc can trigger the conversion of human PrPC into PrPSc,
    suggesting that CWD might be infectious to humans. Interestingly our findings
    suggest that unstable strains from CWD affected animals might not be a problem
    for humans, but upon strain stabilization by successive passages in the wild,
    this disease might become progressively more transmissible to man.


    Our results also have profound implications for understanding the
    mechanisms of the prion species barrier and indicate that the transmission
    barrier is a dynamic process that depends on the strain and moreover the degree
    of adaptation of the strain. If our findings are corroborated by infectivity
    assays, they will imply that CWD prions have the potential to infect humans and
    that this ability progressively increases with CWD spreading.



    Sunday, January 22, 2012

    Chronic Wasting Disease CWD cervids interspecies transmission


    Friday, November 09, 2012

    *** Chronic Wasting Disease CWD in cervidae and transmission to other


    Friday, November 09, 2012

    *** Chronic Wasting Disease CWD in cervidae and transmission to other


    Sunday, November 11, 2012

    *** Susceptibilities of Nonhuman Primates to Chronic Wasting Disease
    November 2012


    Friday, December 14, 2012

    Susceptibility Chronic Wasting Disease (CWD) in wild cervids to Humans 2005
    - December 14, 2012


    kind regards, terry